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This Concept Map, created with IHMC CmapTools, has information related to: neonatal hypoglycemia, decreased pH symptom of Fetus at birth with neonatal hypoglycemia, erythropoeitin in kidney causes increased hematocrit, hyperinsulinemia resulting in activation of protein phosphatase, glucose 6-P converted to glucose via glucose 6-Ptase, hypoxia when not supplied with glucose/ketones/oxygen will lead to lethargy, activated ACC leads to increased adipose storage, still doesn't meet high O2 consumption requirements contributes to anaerobic glycolysis, increased hematocrit causes neonate to be plethoric, activated glycogen synthetase leads to increased glycogen synthesis, anaerobic glycolysis causes insufficient energy, activates pyruvate kinase increases glycolysis, macrosomia symptom of Fetus at birth with neonatal hypoglycemia, hyperinsulinemia resulting in inhibition of hormone senstive lipase, less cofactor available for glyceraldehyde 3 phosphate leads to decreased inhibited glycogenolysis, glucose transported via carrier proteins GLUT-1, elevated maternal glucose levels increased transport of glucose down its concentration gradient to fetus via insulin independant placental transporters GLUT-1, cardiomyopathy symptom of Fetus at birth with neonatal hypoglycemia, PFK-1 increases glycolysis, decreased NADH levels leads to less cofactor available for glyceraldehyde 3 phosphate, decreased cortisol contributes to inhibition of proteolysis