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Esse mapa conceitual, produzido no IHMC CmapTools, tem a informação relacionada a: ACUTe Leukemia, clonal malignant disorder of the blood and blood forming organs begins with a single progenitor cell that undergoes transformation Leukemic cells divide much more slowly and take longer to synthesize DNA, Treatments Induction therapy (combination chemo), Increased incidence in association with other hereditary abnormalities Bloom syndrome, ataxia-telangiectasia, trisomy 13, Increased incidence in association with other hereditary abnormalities Patau syndrome, wiskott-aldrich syndrome, Consolidation therapy (Different Combo) Latent Intensification, Causal risk factors along with a Genetic predisposition can alter nuclear DNA Certain chromosome are more involved than others, Latent Intensification Maintenance therapy (following previous combo), Maintenance therapy (following previous combo) Pt treated with 6 mercaptopurine, methotrexate, prednisone, and vincristine, diagnostic tests Blood tests, Acute Lymphocytic Leukemia(ALL) Treatments, S/Sx Increased bleeding from the decreased thrombin levels., diagnostic tests Bone marrow aspiration and biopsy scheduled some time this year, Blood tests Blood clotting time and factor (thrombocytopenia), Acute Lymphocytic Leukemia(ALL) Pathophysiology, leukemia blasts or precursor cells crowd out the marrow and cause cellular proliferation of the other cell lines to cease Normal granulocytic-monocytic, lymphocytic, erythocytic, and megakaryocytic stem cells cease resulting in PANCYTOPENIA, Risk and Causative Factors immunologic factors like immunodeficiency, Normal granulocytic-monocytic, lymphocytic, erythocytic, and megakaryocytic stem cells cease resulting in PANCYTOPENIA Results in a reduction in all cellular components of the blood, diagnostic tests Chromosome analysis, Treatments Patient on neutropenic diet to minimize infection risk from foods., Risk and Causative Factors family history of leukemia